Download e-book for iPad: Andrology: Male Reproductive Health and Dysfunction by Eberhard Nieschlag, Hermann M. Behre, Susan Nieschlag

By Eberhard Nieschlag, Hermann M. Behre, Susan Nieschlag

ISBN-10: 3540783547

ISBN-13: 9783540783541

This is often the third, absolutely revised variation of a widely known textbook that maintains to symbolize the choicest within the literature on scientific andrology. It examines intensive all points of male reproductive well-being, encompassing the fundamental body structure of male reproductive functionality and quite a lot of problems. all of the chapters is written by way of impressive specialists within the box. one of the subject matters coated are: Testicular functionality, sperm maturation and fertilization, diagnostic systems, infertility, fundamental and secondary hypogonadism, late-onset hypogonadism, erectile disorder, genetic issues, environmental impacts, psychology and sexual medication, testosterone treatment, assisted copy: TESE and ICSI, cryopreservation, male birth control and ethics in andrology.

This quantity will function a textbook for the newcomer to andrology, whereas supplying the skilled doctor with a helpful reference paintings. it's going to entice all who're interested by male reproductive health.

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Additional info for Andrology: Male Reproductive Health and Dysfunction

Sample text

8). Testosterone can act as such or after metabolism to DHT or estradiol. The effects of testosterone and its metabolites appear to vary depending on the experimental model but, in general, we can assume that both T and DHT act mainly at the hypothalamic level by decreasing the frequency of GnRH pulsatility, whereas estrogens depress gonadotropin secretion by reducing the amplitude of LH and FSH peaks at the pituitary level (Hayes and Crowley 1998). Progesterone inhibits gonadotropin release at least in part via arcuate nucleus dopaminergic and NPY neurons (Dufourny et al.

Leptin. Reproductive hormones are inhibited during starvation and kisspeptin mediates some of leptin’s effects on reproduction. According to the current model, leptin and perhaps other adiposity and satiety factors stimulate KISS1 expression, which results in stimulation of GnRH release. When levels of adiposity and satiety factors decrease or when such factors are not detected, the expression of KISS1 (and presumably its secretion) decreases, thus reducing excitatory input to GnRH neurons. Among the other metabolic factors influencing gonadal function, the growth hormone secretagogue ghrelin should be considered as a possible modulator of kisspeptin neurones (Tena-Sempere 2008).

It is therefore believed that the trophic effects of testosterone/FSH on gametogenesis are mediated via somatic Sertoli cells. The testis and the hypothalamo-pituitary system communicate through steroids and protein hormones. Testosterone inhibits the secretion of GnRH and gonadotropins. Inhibin B and follistatin suppress selectively the release of FSH from the pituitary gland, while activin stimulates this process. Beside the effects on gametogenesis, testosterone plays an important role in hair growth, bone metabolism, muscle mass and distribution, secondary sexual characteristics and function of the male reproductive organs.

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Andrology: Male Reproductive Health and Dysfunction by Eberhard Nieschlag, Hermann M. Behre, Susan Nieschlag

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